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Table 1 Baseline and demographic characteristics

From: Obesity drives adipose-derived stem cells into a senescent and dysfunctional phenotype associated with P38MAPK/NF-KB axis

 

Groups

p value

Eutrophic (n = 15)

Obesity (n = 14)

Sex (male), n/total (%)

8/15 (53.3)

4/14 (28.6)

0.1761

Age (years), median (IQR)

29.0 (27.0–37.0)

34.5 (25.0–38.7)

0.7100

BMI, median (IQR)

22.1 (20.7–23.9)

49.3 (43.5–50.6)

< 0.0001

Physical activity, n/total (%)

0/15 (0)

0/14 (0)

–

Comorbidities, n/total (%)

   

 Type 2 Diabetes mellitus

0/15 (0)

1/14 (7.14)

–

 Dyslipidemia

0/15 (0)

4/14 (28.6)

–

 Hepatic Steatosis

0/15 (0)

6/14 (42.8)

–

 Hypertension

0/15 (0)

7/14 (50.0)

–

 Metabolic Syndrome

0/15 (0)

0/14 (0)

–

Medication therapy

   

 ACE Inhibitors (ACEI)

0/15 (0)

3/14 (21.43)

–

 Angiotensin II receptor blockers

0/15 (0)

1/14 (7.14)

–

 Calcium channel blockers

0/15 (0)

1/14 (7.14)

–

 Loop diuretics

0/15 (0)

2/14 (14.28)

–

 Statin

0/15 (0)

1/14 (7.14)

–

 Selective beta-blockers

0/15 (0)

3/14 (21.43)

–

 Thiazide diuretics

0/15 (0)

2/14 (14.28)

–

  1. Abbreviations: ACEI: Angiotensin-Converting Enzyme Inhibitors; BMI: Body mass index; IQR: Interquartile range
  2. Results are shown in frequency (percentage, %) or median (IQR 25–75%)
  3. Results in bold indicate differences between groups, assessed by unpaired t-test (95% confidence interval)